Our principal aim was not to explore the role of EPAS1/HIF2α in neuroblastoma (NB). Neither do we claim to present conclusive data showing that HIF2α is a bona fide NB tumor suppressor. We simply followed our analysis of the transcriptional response induced by 5-aza-deoxycytidine (AZA)+retinoic acid (RA) treatment to the...
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